The range of neurological syndromes associated with glutamic acid decarboxylase antibodies

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Abstract

Neurological syndromes caused by production of antibodies to glutamic acid decarboxylase (GAD65) are a relatively new area of modern clinical neurology, which is of great theoretical and practical interest. High titer of identified antibodies is a not always specific, but highly sensitive, marker for autoimmune CNS disorders. The authors present their own clinical observations and an analysis of the literature on a wide phenotypic range of GAD65-associated pathologies.

 

About the authors

M. Yu. Krasnov

Research Center of Neurology

Email: merritt.kraut@gmail.com
Russian Federation, Moscow

E. V. Pavlov

Research Center of Neurology

Email: merritt.kraut@gmail.com
Russian Federation, Moscow

Margarita V. Ershova

Research Center of Neurology

Email: merritt.kraut@gmail.com
Russian Federation, Moscow

Sofiya L. Timerbaeva

Research Center of Neurology

Email: merritt.kraut@gmail.com
Russian Federation, Moscow

Sergey N. Illarioshkin

Research Center of Neurology

Author for correspondence.
Email: merritt.kraut@gmail.com
ORCID iD: 0000-0002-2704-6282

D. Sci. (Med.), Prof., Corr. Member of the Russian Academy of Sciences, Deputy Director, Head, Department for brain research

Russian Federation, Moscow

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Copyright (c) 2015 Krasnov M.Y., Pavlov E.V., Ershova M.V., Timerbaeva S.L., Illarioshkin S.N.

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